
to everybody from all of us at BCNH.
The college is closed between 24th December 2008 and 4th Janusary 2009. We are open again, as usual, on 5th January 2009.
BCNH Action Plan
1. Next week we will appoint a Scientific Advisor to the college with whom we will have regular meeting and reviews of the course material. We agree there needs to be better scientific rigour to our recommendations.
2. We hope to have a monthly debate with the Scientific Advisor to talk about recent studies, to give us an opportunity for critical self appraisal and to improve our research analysis.
3.
On our blog we should have a regular feature where we review quotes from our own profession, checking for bad science and highlighting spurious claims such as 'bilberries are good for eye strain'!
Deadline for change: 18 November 2009 (as challenged by Ben)
3rd November 2008
Press Release
Nutritional Therapy Council opens discussions on Possible Statutory Regulation of the Nutritional Therapy Profession
The Nutritional Therapy Council (NTC), along with representative of The British Association for Applied Nutrition and Nutritional Therapy (BANT), met the Health Professions Council on 30th October to discuss the possible statutory regulation of practitioners of nutritional therapy.
Ends.
Registered Nutritional Therapist
Nutritional Therapy Council
www.nutritionaltherapycouncil.org.uk
One reason may be toxic metals like:
and other metal poisons that flood the environment and invade your body.
These toxic metals can cause or contribute to a long list of diseases including Alzheimer’s disease, Parkinson’s disease and other brain and neurological disorders. It is worth mentioning that cadmium, a known lung carcinogen, has been recently linked to breast cancer. Strumylaite et all (2008) report that ‘cadmium concentration is significantly higher in malignant breast tissue as compared with normal breast tissue of the same women or benign breast tissue’. Although this was a small study comprising of 21 breast cancer and 19 benign tumour patients, there was a statistically significant difference in cadmium concentration between malignant and benign breast tissues. While the medical establishment recognizes the acute toxicity that comes from high levels of metals in your body, far more people suffer the adverse effects of low-level, chronic exposure.
Mad as a Hatter - How to Avoid Toxic Metals and Clear Them From the Body (PDF)
Dr. Mercola's Comments:
Heavy metal toxicity, just like chemical toxicity, has become one of the most pressing health hazards of our day. Your body is assailed by chemicals and heavy metals on a daily basis, oftentimes from the most innocent-looking sources, from your everyday cookware to your child’s rubber ducky.
Despite these dire warning signs, giant corporations and industry advocates like DuPont and The American Chemistry Council are fighting tooth and nail to block any and all proposed bans on toxic agents that happen to be part of their profit-making schemes. According to EnviroBlog, the latest army of lobbyists is trying to crush
And California State Senate Bill 1713, which would ban BPA from baby bottles and toys, is also under attack by hired guns who are trying to scare everyone into believing this bill will put an end to all canned and jarred goods.
How Do You Know if You’re Suffering from Heavy Metal Toxicity?
Acute heavy metal toxicity is a lot easier to diagnose than that of chronic exposure.
The Weston A. Price diet recommendations, which were written in 1920 and still, like all truths, hold true today, includes these timeless guidelines:
A lot more
You can find their membership form at this link.
Guidelines for Reducing Your Heavy Metal Exposure
Related Articles:
No One is Safe From Chemical Overload
How to Avoid the Top 10 Most Common Toxins
Non-Stick Cookware Continues to Prove Its Toxicity
Our Scientific Advisor, Dirk Budka says:
Before people are being scared regarding heavy metal toxicity I would like to see the studies, which clearly proves the fatality rate and/or severe illnesses in patient.To be a bit cynical… where are all the mercury-triggered Alzheimer patients? Why are dentists who use amalgam for decades still alive?
Of course I am not a friend of amalgam fillings, but I have seen too many patients who removed all amalgam fillings from their teeth and not feeling a bit better.
What about proper toxicity tests (and I mean PROPER tests)?
Just assuming heavy metal toxicity and treating because of symptoms which can be connected to hundreds of diseases is in my opinion very wrong. The author writes:
Avoid cosmetics with aluminium bases, mineral powders that contain bismuth, and aluminium-laden antiperspirants, which have been shown to increase your Alzheimer’s risk by as much as 300 percent! Where is the study? Who did the study? Where was it published? The cross reference in the text leads to MERCOLA.com and one is not able to read anything on this site without subscribing an e-mail letter?!
The author also says “Avoid vaccinations that inject mercury or aluminium directly into your bloodstream.” Again… I wish it would be so easy to follow this advice, which does not give any alternatives.
It is about science and evidence based science. Each and every student must always read the other side. If you believe, that mercury is so bad and you only read the available studies which confirm your opinion… you will be extremely one-sided and in my opinion not a good practitioner. Real science means: look at the other side as well, even if you do not like it.Opinion research is very wrong.
NUTRITIONAL THERAPY COUNCIL
Subject: Regulation of nutrition practitioners.
Statement on behalf of the Nutritional Therapy Council,
the regulatory body for nutritional therapy.
Our national registration scheme will enable members of the public to verify the registration status of individual nutritional therapists.
The National Occupational Standards are set by “Skills for Health”, which is one of the 25 Sector Skills Councils which make up the “Skills for Business” Network. “Skills for Health” is licensed by the Secretary of State for Education and Skills in consultation with Ministers in
Nutritional Therapy Council
http://www.nutritionaltherapycouncil.org.uk/
e-mail ppr@ nutritionaltherapycouncil.org.uk
This article is from NUTRAingrediets.com I Europe.
Almonds demonstrate prebiotic potential
By Shane Starling
1-Jul-2008
Almonds are known for their high vitamin E and mineral content and cholesterol-reducing and diabetes-combating potential but new research indicates they have a prebiotic effect.
Funded by the Almond Board of California (this suggests the research may be biased), the researchers found that almond powder significantly increased levels of certain gut flora. As a result prebiotic effects were increased. My question is why almond powder? Would not eating almonds have the same effect?
The study, published in the July edition of Applied and Environmental Microbiology, found the prebiotic effect did not occur when the fat content was removed from the almond preparation, indicating lipid concentration is key to activating the prebiotic reaction. Almonds, as we know, contain Essential Fatty Acids (EFAs), which we know help beneficial bacteria adhere to the gut wall and help them colonise (90% of the fats in almonds are unsaturated)
"We investigated the potential prebiotic effect of almond seeds in vitro using mixed faecal bacterial cultures," the researchers wrote.
Digested almonds were then mixed with an in vitro batch system to mimic the bacterial fermentation in the large intestine. The populations of intestinal bacteria were then monitored.
"The results show a prebiotic effect of finely ground almonds (with lipid) but not with defatted almonds," Mandalari said. That’s why we need to eat foods that have not been tempered with!
For the finely ground almonds with lipids, the study recorded a significantly increased population for bifidobacteria and Eubacterium rectale, "resulting in a higher prebiotic index (4.43) compared with the commercial prebiotic fructo-oligosaccharides (4.08) at 24h incubation." This finding is of great importance as effects of the gut microbiota for human health are increasingly recognized.
No such effect was recorded for the lipid-free almonds.
"The increase in the numbers of Eubacterium rectale during fermentation of finely ground almonds correlated with increased butyrate production," (butyrate feeds colonocytes) the researchers wrote. "In conclusion, we have shown that addition of finely ground almonds altered the composition of gut bacteria stimulating the growth of bifidobacteria and Eubacterium rectale."
Other research has indicated lipid content of almonds is reduced if almonds are not processed by grinding as in this case, or via mastication. This supports the need for chewing the food properly!!!
The researchers noted the length of time the almond spends in the digestive system also affected the available quantity of lipids and proteins.
They called for further research into almond digestibility, with prebiotic effects tested on human subjects.
Source: Applied and Environmental Microbiology
July 2008, published online ahead of print 23rd May 2008 doi:10.1128/AEM.00739-08
Investigation of the potential prebiotic properties of almond (Amygdalus communis L.) seeds.
Authors: G. Mandalari, C. Nueno-Palop, G. Bisignano, M. S.J. Wickham, and A. Narbad.
Recent research by Jenkins et al (2008) has shown that almonds reduced 24-hour insulin secretion and may in the longer term explain the association of nut consumption with reduced CHD risk. Almonds are also high in protein and fibre, which m ay explain the reduced insulin secretion. Other benefits of almonds are nutrient content such as calcium, magnesium, potassium, vitamin E, and other antioxidants.
Jenkins DJ, Kendall CW, Marchie A, Josse AR, Nguyen TH, Faulkner DA, Lapsley KG, Singer W. (2008) Effect of almonds on insulin secretion and insulin resistance in nondiabetic hyperlipidemic subjects: a randomized controlled crossover trial. Metabolism. Jul;57(7):882-887
Vitamin D is both a vitamin and a hormone. The role of Vitamin D - the ‘sunshine’ vitamin, as we know it appears to have ubiquitous role. Vitamin D in calcium absorption in the gut and normal mineralization of bone is well established. However, research is now focusing on other functions of vitamin D such as innate immune regulation (Hewison 2008), low mood and cognitive performance (Wilkins et al 2006), depression (Berk et al 2007), Metabolic Syndrome prevention (Hyppönen et al 2008), and cancer prevention – see article below.
This article is copied from NUTRAingredietns.com/europe website
http://www.nutraingredients.com/news/ng.asp?n=85255&c=K4kaXXjFbphCJNkBqUaYrw%3D%3D
Prostates protected by vitamin D: study
14-May-2008
The benefits of vitamin D for prostates may be due to the action of the vitamin on a specific gene, suggests new research that deepens our understanding of how nutrients and genes interact.
Researchers from the University of Rochester Medical Center report that the active form of vitamin D in the body, 1,25-hydroxylvitamin D3 (1,25(OH)2D), may link with a gene known as G6PD, which releases an antioxidant enzyme and protect DNA from damage.
"Many epidemiological studies have suggested the beneficial properties of vitamin D," said lead researcher Yi-Fen Lee. "Our findings reflect what we see in those studies and demonstrate that vitamin D not only can be used as a therapy for prostate cancer, it can prevent prostate cancer from happening."
The study is published in the International Journal of Cancer.
The link between vitamin D intake and protection from cancer dates from the 1940s when Frank Apperly demonstrated a link between latitude and deaths from cancer, and suggested that sunlight gave "a relative cancer immunity."
Vitamin D refers to two biologically inactive precursors - D3, also known as cholecalciferol, and D2, also known as ergocalciferol. Both D3 and D2 precursors are hydroxylated in the liver and kidneys to form 25- hydroxyvitamin D (25(OH)D), the non-active 'storage' form, and 1,25-dihydroxyvitamin D (1,25(OH)2D), the biologically active form that is tightly controlled by the body.
There is growing evidence that 1,25(OH)2D has anticancer effects, but the discovery that non-kidney cells can also hydroxylate 25(OH)D had profound implications, implying that higher 25(OH)D levels could protect against cancer in the local sites.
The new study, supported by a grant from the U.S. Department of Defense, appears to provide further compelling evidence of the potent anti-cancer benefits of 1,25(OH)2D.
Normal healthy prostate cells (BPH-1 and RWPE-1) and prostate cancer cells (CWR22R and DU 145) were subjected to oxidative stress. The researchers found that 1,25(OH)2D induced G6PD (glucose-6-phosphate dehydrogenase), a key antioxidant enzyme, in the healthy but not cancer cells. G6PD scavenges reactive oxygen species (ROS) associated with DNA damage.
"If you reduce DNA damage, you reduce the risk of cancer or aging," said Lee said. "Our study adds one more beneficial effect of taking a vitamin D supplement. Taking a supplement is especially important for senior citizens and others who might have less circulation of vitamin D, and for people who live and work areas where there is less sunshine."
"In this study, we have demonstrated that 1,25-(OH)2D can protect nonmalignant human prostate epithelial cells against H2O2-induced cell death through modulating the ROS defense systems, suggesting a possible role of 1,25-(OH)2D in prostate cancer prevention," stated the researchers in the journal.
Vitamin D - bad for cancers, good for you
Lee and co-workers noted that 1,25(OH)2D may act both as an antioxidant and a pro-oxidant, depending on the cellular environment, with a subtle pro-oxidant activity found in cancer cells, while it appears to behave as an antioxidant in normal healthy cells.
"[Our] data suggest that vitamin D might exert a subtle oxidative stress, which could stimulate the detoxification mechanisms to protect cells from the subsequent stress challenges; yet cancer cells lose the 1,25-(OH)2D-induced detoxification responses, therefore, 1,25-(OH)2D acts as a pro-oxidant in cancer cells," they said.
"Our results provide one mechanism to explain how 1,25-(OH)2D protects nonmalignant human prostate epithelial cells from oxidative stress to attenuate the accumulation of oxidative damages during the life.
"Therefore, vitamin D might be beneficial for preventing the development of age-dependent diseases," they concluded.
Source: The International Journal of Cancer
122, 2699-2706 (2008)
"Protective role of 1a, 25-dihydroxyvitamin D3 against oxidative stress in nonmalignant human prostate epithelial cells"
Authors: B.-Y. Bao, H.-J. Ting, J.-W. Hsu, Y.-F. Lee
British scientists Elina Hyppönen and Chris Power (2007) from the
Interestingly, other studies have also shown that sunshine levels in some northern countries are so weak during the winter months that the body makes no vitamin D at all, resulting in over half of the population to have insufficient or deficient levels of the vitamin.
So - how much vitamin D should we take?
While Committee on Medical Aspects of Food and Nutrition Policy (COMA) have not established a RNI for vitamin D, estimated maximum daily intake is 22mcg (800iu). A review by Talwar et al (2007) reported that the tolerable upper intake level for oral vitamin D3 should be increased to 250 micrograms per day (10,000 iu), which is a 12.5 x increase.
Dietary sources of vitamin D
Oily fish such as salmon, mackerel, tuna and sardines are the richest source of vitamin D. However, smaller amounts are also found in foods such as milk, fortified margarine, fortified cereals, eggs, beef liver & Swiss cheese.
Sun exposure – free vitamin D
It has been suggested that approximately 5 - 30 minutes of sun exposure between 10am and 3pm at least twice a week, to the face, arms, legs, or back, without a sunscreen, can usually lead to sufficient vitamin D synthesis. Moderate use of commercial tanning beds that emit 2 - 6% UVB radiation may also be effective
(Wolpowitz and Gilchrest 2006). They also recommend that individuals with limited sun exposure need to include good sources of vitamin D in their diet or take a supplement.
However, they equally caution to limit exposure of skin to sunlight as UV radiation is a carcinogen responsible for most of skin cancers and deaths from metastatic melanoma. Lifetime cumulative UV damage to skin may also be largely responsible for some age-associated dryness and premature ageing of the skin.
References:
Berk M Sanders KM Pasco JA Jacka FN Williams LJ Hayles AL Dodd S (2007) Vitamin D deficiency may play a role in depression. Medical Hypotheses May 10; [Epub ahead of print]
Hewison M (2008) Vitamin D and innate immunity Current Opinion in Investigational Drugs May 9(5):485-90
Holick MF (2007)Vitamin D deficiency. The
Hyppönen E Boucher BJ Berry DJ Power C (2008) 25-hydroxyvitamin D IGF-1 and metabolic syndrome at 45 years of age: a cross-sectional study in the 1958 British Birth Cohort. Diabetes Feb 57(2):298-305
Hyppönen E Power C (2007) Hypovitaminosis D in British adults at age 45 y: nationwide cohort study of dietary and lifestyle predictors. The American Journal of Clinical Nutrition Mar;85(3):860-8
Talwar S A Aloia J F S Pollack and Yeh JK (2007) Dose response to vitamin D supplementation among postmenopausal African American women The American Journal of Clinical Nutrition December 86 (6):1657-1662
Wilkins CH Sheline YI Roe CM Birge SJ Morris JC (2006) Vitamin D deficiency is associated with low mood and worse cognitive performance in older adults. The American Journal of Geriatric Psychiatry Dec 14(12):1032-40.
Wolpowitz D Gilchrest BA (2006) The vitamin D questions: how much do you need and how should you get it? Journal of the